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Scientific Program
6th International Conference on Aging & Gerontology , will be organized around the theme “Theme: Advancing in Aging Care Management and Exploring Therapeutic Opportunities”
AGING CONFERENCE 2023 is comprised of 17 tracks and 0 sessions designed to offer comprehensive sessions that address current issues in AGING CONFERENCE 2023.
Submit your abstract to any of the mentioned tracks. All related abstracts are accepted.
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The aging process is driven at the cellular level by random molecular damage that slowly accumulates with age. Although cells possess mechanisms to repair or remove damage, they are not 100% efficient and their efficiency declines with age.
Genetic mutations occur and accumulate with increasing age, causing cells to deteriorate and malfunction. In particular, damage to mitochondrial DNA might lead to mitochondrial dysfunction. Therefore, aging results from damage to the genetic integrity of the body's cells.
During aging, many tissues show a decline in regenerative potential coupled with a loss of stem cell function. Cells including somatic stem cells have evolved a series of checks and balances to sense and repair cellular damage to maximize tissue function.
Apoptosis and senescence are two types of cellular response to damages that are altered in both cancer and aging, albeit through different mechanisms. Carcinogenesis is associated with a progressive reduction in the ability of the cells to trigger apoptosis and senescence.
Aging refers to the gradual loss of cellular function, and regeneration is the repair of damaged tissue generally mediated by preexisting adult or somatic stem cells. Scientists are interested in understanding the biological mechanisms behind these two complex processes.
The process of aging refers to the decay of an organism’s structure and function, in which molecular and cellular modifications can have various effects at the individual level over the course of a lifetime. The accumulation of molecular errors that compromise adult stem cell functions occurs because of genetic and epigenetic interactions and depends on hereditary, environmental, and stochastic factors. Here we review the known genetic factors involved in aging.
Genome instability has long been implicated as the main causal factor in aging. Somatic cells are continuously exposed to various sources of DNA damage, from reactive oxygen species to UV radiation to environmental mutagens.
Cognitive impairment manifests in various psychiatric disorders such as schizophrenia, bipolar disorder, depressive disorder, attention deficit/hyperactivity disorder (ADHD), post-traumatic stress disorder (PTSD), and obsessive-compulsive disorder (OCD) (4).
In the case of transcriptional regulation, a single gene can be regulated during the conversion of DNA to pre-mRNA in a range of ways, for example from altering the number of copies of RNA that are transcribed, to the control of transcription factors when the gene is transcribed.
Use of high-density oligonucleotide arrays representing 6347 genes revealed that aging resulted in a differential gene expression pattern indicative of a marked stress response and lower expression of metabolic and biosynthetic genes.
Social gerontology is seen as a science-based but application-oriented subdiscipline of gerontology. It focuses particularly on social relationships in old age, social participation of elderly and old people and the protection of their individual needs. Self-determination and autonomy are important value orientations.
Somatic Disease and Multiple Chronic Conditions
Cardiovascular Disease. Cardiovascular disease remains the most common cause of death of older adults, although death rates have dropped in the last 20 years. ...
Cancer.
Diabetes Mellitus.
Osteoporosis.
Multiple Chronic Conditions.
Too often the elderly are invisible to others, and perhaps especially to teenagers. As adults, we can teach students to show respect and to demonstrate compassion towards the elderly by giving students opportunities to better understand the impact of discrimination or apathy toward the elderly. We can also help students develop empathy and practice compassion and respect for the elderly in their day-to-day lives.
The following case study includes a short story from multiple viewpoints and a set of questions designed to facilitate discussion about respecting and caring for the elderly and the importance of maintaining commitments and volunteering for selfless reasons.
Thinking about joining a clinical trial? Clinical research has led to the discovery of every disease treatment prescribed today. Read these articles to learn the benefits and risks of participating in a clinical study and how they work. By being part of a clinical trial, you may help others, including future generations, get better treatments.
Obsession with a youthful appearance has become commonplace in modern society and has resulted in an upswing in cosmetic procedures trying to reverse the aging process. We selectively review the literature on aging and cosmetic surgery, with particular regard for the aging face. We pay attention to psychosocial aspects of response to such cosmetic procedures, both in terms of outcome and with respect to risk factors for a poor outcome.
The goal of remediation is to identify, based on course instructional objectives and evidenced by poor performance on examination(s), areas of weaknesses of material, and, once identified, to assist the student in overcoming those weaknesses and develop mastery of the material.
Aging is a physiological process mediated by complex pathways and driven by various acquired and genetic factors. To clarify the reciprocal connection of the aging process and the deterioration of stem cell function is crucial, not only to understand the pathophysiology of the disorders associated with aging, but also to make possible the development of new therapeutic interventions to slow down, and possibly reverse, the age-related degenerative processes. In the present chapter, we aim to give an overview on the potential therapeutic approaches for the treatment of aging-induced stem cell dysfunction, focusing on the principal extracellular and cellular mechanisms responsible for stem cell aging.